ICU Map

ICU hemostasis

TEG / ROTEM Lab

Learn to move from trace shape to a clinical phenotype, then to a targeted and locally validated action. The schematics are teaching aids—not patient results or universal transfusion thresholds.

Paired-channel interpretation

TEG / ROTEM Bedside Lab

Read the two channels together: identify the dominant phenotype, confirm it in the patient, and avoid treating a trace in isolation.

Representative teaching traces · not a patient result

Worked interpretation

Low fibrin-based firmness during major bleeding

A bleeding trauma or obstetric patient has weak FIBTEM firmness relative to the total-clot channel.

EXTEM

Total clot

Teaching readout

Clot builds, but final firmness is reduced

CT
not the dominant problem
A5
reduced
MCF
reduced

FIBTEM

Fibrin contribution

Teaching readout

Fibrin-based firmness is disproportionately low

A5
markedly reduced
A10
markedly reduced
MCF
low

Dominant phenotype

A fibrinogen deficit or impaired fibrin polymerisation is a leading phenotype—not a diagnosis by itself.

Confirm before acting

Check clinical bleeding, Clauss fibrinogen and the local FIBTEM/functional-fibrinogen algorithm; correct calcium, temperature and major dilution at the same time.

Do not conclude

Do not give platelets alone or copy a cryoprecipitate/fibrinogen dose from another device protocol.

60-second reading order: confirm clinically important bleeding and source control → check initiation → compare early firmness (A5/A10 where used locally) → compare total and fibrin-specific channels → check lysis or heparinase comparison → intervene through the local pathway and repeat the clinical/trace assessment.
Approximate parameter mapping between TEG and ROTEM
TEGROTEMTeaching interpretation
TEG R-timeROTEM CTInitiation: factor deficiency, anticoagulant effect, dilution or pre-analytic problems are possible contributors.
TEG K / alpha angleROTEM CFT / alpha anglePropagation: use with fibrinogen assessment and local reagent-specific ranges.
TEG MAROTEM MCFMaximum firmness: combines platelet and fibrin contributions.
TEG LY30ROTEM ML / CLILysis: timing and cut-offs differ by analyser and protocol.

Trace shapes, labels and readouts are teaching schematics and not a universal trigger. Numeric reference intervals, assay availability and treatment triggers depend on the instrument, reagent, patient population and local validated algorithm.